![]() ![]() Millen SJ, Daniels D, Meyer G (1990) Gadolinium-enhanced magnetic resonance imaging in facial nerve lesions. Gardner G, Robertson JH (1985) Facial nerve function in cerebellopontine angle tumor surgery. Glasscock ME III, Hayes JW, Jackson CG, Steenerson RL (1978) A one-staged combined approach for the management of large cerebellopontine angle tumors. Hearing loss resulting from cerebellopontine angle tumor is most commonly caused by vestibular schwannomas, which arise directly from the sheath of the vestibular nerve (VIII) in the internal auditory canal. Am J Otol 10: 174–176Ĭohen NL, Ransohoff J (1984) Hearing preservation - posterior fossa approach. The lesion was nearly 4 cm in maximum dimension and extended into the internal auditory canal. Valvassori GE, Guzman M (1989) Growth rate of acoustic neuromas. Radiology 174: 93–98īurke JW, Podrasky AE, Bradley WG (1990) Meninges: benign postoperative enhancement on MR images. AJNR 9: 27–34Įlster AD, DiPersio DA (1990) Cranial postoperative site: assessment with contrast-enhanced MR imaging. Lanzieri CF, Larkins M, Mancall A, Lorig R, Duchesneau PM, Rosenbloom SA, Weinstein MA (1988) Cranial postoperative site: MR imaging appearance. perpendicular to the plane of the internal auditory canal.7. Glasscock ME, Shambaugh GE, Johnson GD (1990) Surgery of the ear. Keywords: Sensorineural hearing loss, imaging, CT, MRI. Glasscock ME, Kveton JF, Jackson SC, McKennan KX (1986) A systematic approach to the surgical management of acoustic neuroma. Mueller DP, Gantz BJ, Dolan KD (1992) Gadolinium-enhanced MR of the postoperative internal auditory canal following acoustic neuroma resection via the middle fossa approach. In group 4, grafts may prevent adequate visualization of the IAC. In group 3 residual or recurrent tumor cannot be excluded. Whether follow-up in these groups is indicated needs to be determined. In groups 1 and 2, the MRI features correlate well with complete tumor removal. In group 4, follow-up in 1 of the 2 patients was stable. In group 3 follow-up showed 1 tumor recurrence (surgically confirmed) and 4 stable abnormalities. In group 2, dural enhancement remained unchanged in 5 patients and decreased in 3. Prospective 1-to 2-year follow-up studies were available in 8, 5, and 1 patients in groups 2, 3, and 4 respectively. We found four patterns (1) internal auditory canals (IAC) with nonenhancing soft-tissue strands, possibly scars or distorted residual nerves (8) (2) IAC with marginal enhancement-reactive dura mater (16) (3) IAC with contrast-enhancing globular tissues suggesting residual or recurrent tumour (5) (4) high-signal intensity in the IAC before contrast medium administration, probably related to graft with fat/fascia/muscle (2). The internal acoustic canal (IAC), also known as the internal auditory canal or meatus (IAM), is a bony canal within the petrous portion of the temporal bone that transmits nerves and vessels from within the posterior cranial fossa to the auditory and vestibular apparatus. Follow-up MRI was performed after 1–2 years on patients with questionable abnormalities. Prospective baseline MRI was obtained on 31 patients who had “total” removal of acoustic schwannoma 6 months to 9 years previously. ![]()
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